Adenovirus-mediated gene transfer of superoxide dismutase and catalase decreases restenosis after balloon angioplasty

Eric Durand*, Ayman Al Haj Zen, Faouzi Addad, Camille Brasselet, Giuseppina Caligiuri, François Vinchon, Patricia Lemarchand, Michel Desnos, Patrick Bruneval, Antoine Lafont

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

31 Citations (Scopus)

Abstract

Background: Reactive oxygen species (ROS) production increases after injury and potentially contributes to restenosis after angioplasty. We therefore evaluated the effect of adenovirus-mediated gene transfer (Ad) of superoxide dismutase (SOD) and catalase (CAT) on ROS production and restenosis after balloon angioplasty. Methods: O2- and H2O 2 production was quantified in cultured cells after incubation with either LPS or CuSO4. Angioplasty and gene transfer were performed in rabbit atherosclerotic iliac arteries. One artery was injected with AdSOD and AdCAT, while the contralateral artery was injected with an adenovirus carrying no transgene, and served as control. Results: ROS production was significantly decreased after adenovirus-mediated gene transfer of SOD and CAT as compared with control. Treated arteries showed less restenosis (32 ± 27 vs. 63 ± 19%, p = 0.003) and less constrictive remodeling (1.2 ± 0.3 vs. 0.9 ± 0.2, p = 0.02) than control arteries. Arteries injected with AdSOD and AdCAT showed better vasoreactivity to acetylcholine (11 ± 4 vs. -1 ± 6%, p < 0.05), lower collagen density (43 ± 16 vs. 53 ± 23%, p = 0.03), and lower inflammatory cell infiltration (22 ± 6 vs. 36 ± 11%, p = 0.04) than control arteries. Conclusions: Our data suggest that adenovirus-mediated gene transfer of SOD and CAT reduced oxidative stress, restenosis, collagen accumulation, and inflammation and improved endothelial function after angioplasty.

Original languageEnglish
Pages (from-to)255-265
Number of pages11
JournalJournal of Vascular Research
Volume42
Issue number3
DOIs
Publication statusPublished - May 2005
Externally publishedYes

Keywords

  • Endothelial function
  • Extracellular matrix
  • Gene therapy
  • Redox signaling
  • Restenosis

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